
Michael Morales, associate professor of physical science, makes a proposal for the intellectual property bill during the Faculty Senate meeting in the Kanza room Tuesday afternoon. Morales pushed for this bill to pass, which would give the university’s professors recognition for their creative property. Julie Thephachan/The Bulletin
The amendment of a Faculty Senate bill regarding the intellectual property policy was the focus of debate at the meeting on Tuesday in Webb 2 Lecture Hall.
“The reason I put (the amendment) together is because this doesn’t reflect a good balance between the university’s rights and the faculty member’s rights who might create it (an intellectual property),” said Michael Morales, associate professor of physical sciences department.
Morales said some of the original policies, like “the university shall have the right to revise (mediated courseware) and decide who will utilize the mediated courseware,” were fairly strong.
“What I tried to do then was to go through (the amendment) and make it more balanced,” Morales said. “For example, in most places where the university was deciding an issue, I thought that should be mutually agreed upon by the creator or creator’s courseware and the university.”
Marian Riedy, assistant professor of business administration and education, said she agreed with some of the amendment, but thought there was some confusion remaining about the meaning and intent of the proposed policy.
“I think the amendment was directed to the part of the policy dealing with delivery methodology,” Riedy said, “but I think the main concern, which is addressed in the proposed policy, is the ownership of course materials, which is in another part of the policy.”
She said she was also concerned that some parts of the amendment might conflict with the Kansas Board of Regents’ intellectual property policy.
Two motions were made. One passed 19-6 deciding that the amendment should be separated into four specific pieces. The other passed 20-6 that the amendment should be tabled until the next meeting on Nov. 15.
“A rush vote is too dangerous to me,” said Deborah Gerish, associate professor of social sciences department. “Take some time, let people understand what the amendment really means. My department tells me what they want me to vote. Then we are all good.”
The presidential search also brought to attention the lack of available candidate information and the limited time faculty and students have to provide feedback.
“The Board of Regents has a different view on the campus,” said Marvin Harrell, professor of mathematics and chair of Faculty Affairs. “If we are the sellers rather than the buyers…we really aren’t looking at them – they are looking at us. At least that’s the impression that’s been given, and it’s the problem we need to be thinking about and then we expect the senate might want to consider a resolution.”
The Phoenix Charity Foundation is a 501(c)(3) non-profit organization, which exists to support the families of international students in emergency situations. It was founded and acquired its IRS certification within a week after the fire that killed two ESU students over fall break, by Sisi Huang, president of the Chinese Student Association and business manager for The Bulletin.
“What amazed me was that it’s a 501 (c)(3) registered organization, and she got it done in a week, which is unheard of,” said Kevin Johnson, Faculty Senate president. “I don’t know how she did it. It almost made me wish I had a company so I could offer her a job.”
Two other bills regarding credit hours for course and a time limit we on their second reading. Both were passed unanimously and forwarded on to interim President H. Edward Flentje for approval.
The next meeting will be at 3:30 p.m. on Nov. 15 in Webb 2 Lecture Hall.
Tianhai Jiang
Langerhans cell histiocytosis.(PAHTHOLOGY CLINIC)(Report)
Ear, Nose and Throat Journal March 1, 2010 | Camelo-Piragua, Sandra Langerhans cell histiocytosis (LCH; formerly histiocytosis X) is a neoplastic proliferation of Langerhans cells (antigen-presenting histiocytes). Involvement of osseous and extraosseous sites of the head and neck has been reported in as many as one-third of cases. Osseous LCH may involve the flat bones of the skull, the facial bones, the bones of the jaw and sinonasal tract, and the medial part of the external auditory meatus. Destructive bone lesions can manifest as headache, toothache, tooth loss, hearing loss, and otitis media. Involvement of the skull can also cause exophthalmos and diabetes insipidus.
[FIGURE OMITTED] Radiographically, bone lesions appear as sharp, punched-out radiolucencies. Sites of extraosseous involvement include the facial skin and scalp, the periorbital region, the gingiva, and the cervical lymph nodes. see here langerhans cell histiocytosis
Demographically, LCH occurs mainly in children (~1/200,000 annually), it is rare in blacks, and it has a predilection for males (male-to-female ratio 3.7:1).
It is important to remember that while LCH may present as a solitary lesion (known as an eosinophilic granuloma), it can also be multifocal and involve several systems (e.g., the liver, spleen, lung, gastrointestinal tract, and central nervous system). In Hand Schuller-Christian disease, there is multifocal involvement of a single tissue, usually bone. In Letterer-Siwe disease, there is multisystem involvement. Patients can also present with fever, rashes, and pancytopenia. The etiology of LCH is unknown.
On tissue biopsy, the histopathology of LCH is distinctive. Lesions are characterized by an accumulation of mononuclear and multinucleated Langerhans cells admixed with abundant mature eosinophils (figure, A), as well as some neutrophils and small lymphocytes. Eosinophilic abscesses can feature central necrosis. Langerhans cells have grooved, folded, indented, or lobulated vesicular nuclei (figure, B). They are typically positive for immunohistochemical stains against CDla (figure, C), langerin, S-100 protein, and CD68. The hallmark of Langerhans cells is the ultrastructural presence of cytoplasmic Birbeck granules, which are rod- or tennis-racket-shaped structures (figure, D). LCH must be distinguished from reactive histiocytosis, Hodgkin lymphoma, NK/T-cell lymphoma, ErdheimChester disease (a CD 1 a-negative histiocytic disorder), and Rosai-Dorfman disease.
Patients with treated unifocal LCH have an excellent prognosis, but the presence of multisystem in volvement is a poor prognostic sign. Unifocal disease progresses to multisystem disease in about 10% of patients. Spontaneous regression has occurred in rare cases. The choice of treatment depends on the number of sites involved. Solitary lesions, which frequently occur in the head and neck, can be conservatively resected with curettage or excision. Systemic chemotherapy is administered to patients with disseminated or multifocal disease and to those who do not respond to local treatment. website langerhans cell histiocytosis
Suggested reading Buchmann L, Emami A, Wei JL. Primary head and neck Langerhans cell histiocytosis in children. Otolaryngol Head Neck Surg 2006;135(2):312-17.
Davis SE, Rice DH. Langerhans’ cell histiocytosis: Current trends and the role of the head and neck surgeon. Ear Nose Throat J 2004;83(5):340, 342, 344 passim.
Quraishi MS, Blayney AW, Walker D, et al. Langerhans’ cell histiocytosis: Head and neck manifestations in children. Head Neck 1995;17(3):226-31.
From the Department of Pathology, Massachusetts General Hospital, Harvard Medical School, Boston (Dr. Camelo-Piragua); the Department of Pathology, Medical College of Wisconsin, Milwaukee (Dr. Zambrano); and the Department of Pathology, Baystate Medical Center, Tufts University School of Medicine, Springfield, Mass. (Dr. Pantanowitz).
Camelo-Piragua, Sandra